The Day Last Friday I had the amazing [...]
"This reflective writing piece was completed by Dr. Aashish Kalani [...]
Rob Arntfield, MD, FRCPC and Atul Jaidka, MD, PGY2 [...]
This week is a case of a 29 yo F with known severe pulmonary hypertension. The etiology was thought secondary to cocaine-induced idiopathic pulmonary arterial hypertension. Unfortunately, she sustained a cardiac arrest (you’ll see why when you look at the images). ROSC was obtained and she was transferred to the ICU. Despite maximal medical support of her RV (optimal ventilator management, IV flolan, inhaled nitric oxide, inotropes including milrinone and vasopressin) she had persistent hypotension and worsening renal failure necessitating CRRT. The overnight team decided to trial some small boluses of crystalloid to see if that would help. Have a look at the images below and decide whether or not you would give fluids or recommend something else? If I said the CVP (as measured from the right IJ central line) was 22, what would the estimated RVSP be?
It’s a 78 yo M with unwitnessed syncope, a subsequent tib-fib fracture, who was eventually admitted to the ICU for persistent hypotension and altered LOC that had not been fully elucidated. He had an extensive work up including a negative CTPA, CT head, and ultimately even an angiogram (based on some transient diffuse ST depression and a positive troponin) which showed clean coronary arteries. He eventually stabilized with good supportive care, and the ICU team was now trying to wean him off the ventilator and were aggressively diuresing him. They asked for a POCUS assessment to help guide further volume management. Have a look at the images. What two major findings are most striking? Should the team continue to diurese him or perhaps give some volume back?
This is a 39 yo F post cardiac arrest NYD. She was in the weaning stages of her care and close to extubation, but she became newly febrile with increasing oxygen requirements. Her sputum culture was positive for E. coli. A portable CXR was done which did not show any obvious large consolidations. A POCUS thoracic study was performed. An unusual finding was seen on the left side (shown in the clips below). Also, to orient those who aren't familiar with the WesternSono shorthand here is a legend for the labels: L1 = Left anterior chest wall, L2 = Left anterior axillary line, L3 = Left costophrenic view, and L4 = Left PLAPS (PosteroLateral Alveolar and/or Pleural Syndrome)
This year marked the 10th anniverary for the annual Canadian [...]
This is 54 yo M who presented for an elective surgery. On POD # 0 he became tachycardic with subjective dyspnea and hypotension progressing into a PEA arrest. ROSC was quickly obtained with typical ACLS and he was placed on life support and transferred to the ICU for further management. A CTPA was negative, and his EKG was unremarkable with no evidence of coronary ischemia. On POD # 1 he remained hemodynamically unstable, and given very poor transthoracic windows, the decision was made to perform a TEE. Have a look at the images and Doppler information below. Is there a finding that may explain the etiology of his arrest? What would your recommendation be to the treating team?
This is a 35 yo M PWID who presented with a right septic AC joint, MRSA bacteremia and hypoxic respiratory failure. He was taken to the OR for washout of his AC joint and subsequently transferred to the ICU for post-op management. A post-operative CXR showed some patchy consolidations but no obvious pleural effusions. The POCUS team was subsequently deployed. Interrogation at the costophrenic angle and PLAPS (posteroalveolar and/or pleural syndrome) point on both sides yielded the following images. What do you see and what should the next steps in management be?